refraction 发表于 2025-3-30 08:37:04
Hanane Djellab,Amel Bouchemha,Fouzia Maamri,Farouk Boumehrez,Abdelhakim Sahourppropriate therapy. This textbook, the first of its kind, addresses all aspects of the disorder – from genetics, pathophysiology, and ongoing research,to clinical presentations, risk factors, and treatment..978-3-030-22096-9978-3-030-22094-5针叶 发表于 2025-3-30 15:06:09
http://reply.papertrans.cn/47/4660/465910/465910_52.png使服水土 发表于 2025-3-30 18:18:05
http://reply.papertrans.cn/47/4660/465910/465910_53.png考古学 发表于 2025-3-30 23:18:21
Omar Bouagila Algaderi,Abdulkhalek M. Zatoutsponsible for the frequently fatal multiorgan system failure see in MAS. Whole-exome sequencing as well as targeted sequencing of pHLH-associated genes in patients with sJIA-associated MAS, demonstrated increased “burden” of rare protein altering variants affecting the cytolytic pathway compared to无能的人 发表于 2025-3-31 01:47:22
http://reply.papertrans.cn/47/4660/465910/465910_55.png退潮 发表于 2025-3-31 07:22:55
http://reply.papertrans.cn/47/4660/465910/465910_56.pngbeta-cells 发表于 2025-3-31 10:46:48
http://reply.papertrans.cn/47/4660/465910/465910_57.png杀虫剂 发表于 2025-3-31 13:26:25
Alharari Alsouri Alharari,Sami Saddek Bizzan,Abdulmoied Omar cancer stem cell (CSC), the CXCL8–CXCR1/2 signaling might be a key player in tumor development and metastasis. Agents targeting CXCR1/2 and CXCL8 antibodies have been discovered over past 20 years. Such inhibitors are anticipated to be effective in a variety of inflammatory disorders when used alon哑巴 发表于 2025-3-31 18:29:35
ells, or astrocytes, which are able to present myelin antigens in the context of their MHC class II molecules. In this inflammatory microenvironment, T lymphocytes and other cells express cytokines and chemokines, which both promote and regulate the pathogenic process.Neutropenia 发表于 2025-4-1 01:35:31
Petar Bisevac,Ana Toskovic,Mohamed Salb,Luka Jovanovic,Aleksandar Petrovic,Miodrag Zivkovic,Nebojsa ectively, these observations suggest that a significant number of autoreactive T cells ordinarily escape both negative selection in the thymus and clonal deletion in the periphery, and that their survival in the peripheral pool of mature T cells is not, in and of itself, predictive of autoimmune dis