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Prokaryotic control of transcription: How and why does it differ from eukaryotic control?,llow the bacteria to approach or to flee particular chemicals (Adler, 1975; Boyd and Simon, 1982). We will not discuss such mechanisms here. Other mechanisms adapt the transcription rates of genes whose products are needed or not needed in a particular environment. Genes which deal with the cataboliDAMP 发表于 2025-3-25 17:15:43
The Heat Shock Transcriptional Response,onses. The response to temperature elevation, exposure to toxic agents, or other physiological stresses is universal and mediated through the induction of a highly conserved set of genes referred to as heat shock (HS) genes (Lindquist, 1986; Lindquist and Craig, 1988; Morimoto and Milarski, 1990; MoConflagration 发表于 2025-3-25 20:14:47
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,NF-κB: A Mediator of Pathogen and Stress Responses,ntal challenges. Bacteria, viruses, parasites, injury, radiation, oxidative stress, numerous chemical agents and many cytokines released in response to such challenges are all well-studied and potent activators of this transcription factor in a number of different cell types. Activated NF-κB then in暴发户 发表于 2025-3-26 07:02:43
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