Supplement 发表于 2025-3-23 10:32:09
http://reply.papertrans.cn/47/4625/462403/462403_11.pngCommission 发表于 2025-3-23 14:21:17
Xiangyan Ruan,Alfred O. Mueckocrine (digestive) func tions commonly leads to severe gastrointestinal disturbances, malabsorption, a cata bolic state, and weight loss in the face of an adequate diet. Loss of endocrine pancreatic function leads to a large spectrum of disorders associated with the loss of hormone secretions; the为敌 发表于 2025-3-23 19:27:23
http://reply.papertrans.cn/47/4625/462403/462403_13.pngscotoma 发表于 2025-3-23 23:27:32
The Brain Phenotype in Polycystic Ovary Syndrome (PCOS): Androgens, Anovulation, and Gender,ltiple genetic allelic variants that are independently associated with hyperandrogenism, gonadotropin regulation, timing of menopause, depression, and metabolic disturbances, including insulin resistance . Of note, the data cited above showed that not all women with PCOS possess the full compleme灌输 发表于 2025-3-24 05:01:04
http://reply.papertrans.cn/47/4625/462403/462403_15.pngobligation 发表于 2025-3-24 07:16:48
Toward Adolescent Prevention of Adult Anovulation in Polycystic Ovary Syndrome, energy-rich environment. This mismatch results in a sustained requirement to store more fat than is safely feasible in subcutaneous adipose tissue, and the excess of fat ends up being stored in ectopic depots, notably in the liver and viscera (= hepato-visceral or central fat). In adolescent girlsincubus 发表于 2025-3-24 11:33:08
http://reply.papertrans.cn/47/4625/462403/462403_17.png移动 发表于 2025-3-24 17:43:52
Pathogenesis of PCOS: From Metabolic and Neuroendocrine Implications to the Choice of the Therapeutprobably they need an update according to the new insights recently ascertained, that is, insulin resistance (IR) and compensatory hyperinsulinemia. In addition, new specific insights have been demonstrated in animal models of PCOS that suggest a clear role of a neuroendocrinological impairment thatagnostic 发表于 2025-3-24 21:57:50
Impact of Polycystic Ovary Syndrome, Metabolic Syndrome, Obesity, and Follicular Growth Arrest in Wevalence varies between 8% and 13% . This syndrome was initially defined as a reproductive disease characterized by the presence of hyperandrogenism (HA), ovulatory dysfunction, and polycystic ovaries. However, growing data have shown the strong association with metabolic dysfunction. AlthoughMETA 发表于 2025-3-25 01:03:16
http://reply.papertrans.cn/47/4625/462403/462403_20.png