HATCH
发表于 2025-3-28 18:39:24
Cell-mediated Immunity in Glomerulonephritis,ephritis has grown largely from immunohistological studies of human renal biopsies in the 1960s. These studies revealed humoral immune reactants, principally antibody and complement, deposited in the majority of diseased glomeruli. Developments in cellular immunology also allowed . assessment of T l
性冷淡
发表于 2025-3-28 20:48:52
Eicosanoids and Cytokines in Glomerular Injury, of antibody, either directed against trapped or endogenous glomerular antigens, or in the form of immune complexes, usually results in the activation of complement, the accumulation of inflammatory phagocytes and platelets and the modulation of endogenous glomerular cell function. It is now evident
我要威胁
发表于 2025-3-29 00:43:18
Immunology of Minimal-change Nephropathy,here has been very little progress in solving the aetiology and pathogenesis of minimal-change nephropathy (MCN). The central observation of a disorder characterized by an absence of inflammatory changes and immune reactions in the diseased organ, but with a high response rate to drugs which profoun
吸气
发表于 2025-3-29 05:21:56
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难理解
发表于 2025-3-29 08:35:01
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Champion
发表于 2025-3-29 12:34:59
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无可非议
发表于 2025-3-29 16:42:28
Autoimmunity in Systemic Vasculitis, the size and site of vessels involved. Ideally classification should be according to mechanisms of pathogenesis, but until recently so little information was available that classification has been according to morphological criteria (Table 1).
详细目录
发表于 2025-3-29 23:30:13
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Microaneurysm
发表于 2025-3-30 00:44:01
Immunopathogenic Mechanisms of Interstitial Nephritis,g the ultimate prognosis.. In some settings, indices of tubulointerstitial damage display a better correlation with glomerular filtration rate than do indices of glomerular damage.. Such findings have stimulated a growing interest in understanding mechanisms of injury to the tubulointerstitium.
开始没有
发表于 2025-3-30 06:15:13
The Membrane Attack Complex of Complement in Renal Injury,ytic enzymes. In the Arthus reaction (a classical model of inflammation), complement deposits indicating complement activation are seen at the site of antigen-antibody reaction, together with neutrophil infiltration (for review see refs. 1, 2).