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Lilo Endrissncer therapy to provide an apoptotic stimulus when cellular damage induced by the therapeutic agent is detected by the sensor system. Therefore, it is not surprising that failure in pro-survival checkpoint resp978-1-61779-635-7978-1-60761-178-3Series ISSN 2196-9906 Series E-ISSN 2196-9914行业 发表于 2025-3-27 08:12:24
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Book 20151st editionn des Gegenübers oder durch soziale Ausgrenzung. Hier sind Führungskräfte gefordert. Dieses fundierte Buch sensibilisiert den Leser für das Thema und stellt eine Fülle von Methoden zur Verfügung, um Fallen aufzudecken und Betroffene zu stabilisieren..躺下残杀 发表于 2025-3-27 16:38:03
tosis responses depend on conserved DNA damage checkpoint proteins. Genetics- and genomics-based approaches that have demonstrated roles for conserved checkpoint proteins have also begun to uncover novel components of these response pathways. In this chapter, we will briefly review the . DNA damageCRUE 发表于 2025-3-27 21:45:50
Lilo Endrissect p53 target genes. Taqman® analysis and quantitative chromatin immunoprecipitation analysis are used to validate the . prediction and microarray data. Enrichment factor analysis is used to demonstrate that . prediction greatly enriches for genes that are transcriptionally regulated by p53 and ass新奇 发表于 2025-3-28 00:11:30
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Lilo Endrissth rapamycin, an inducer of p27.. Furthermore, Cdh1-deficient DT40 cells fail to maintain DNA damage-induced G2 arrest, and Cdh1-APC is activated by X-irradiation-induced DNA damage. Thus, activation of Cdh1-APC plays a crucial role in both cdk inhibitor-dependent G1 arrest and DNA damage-induced G2遗留之物 发表于 2025-3-28 07:59:23
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