轻舟
发表于 2025-3-21 18:44:08
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神圣不可
发表于 2025-3-21 22:56:18
phosphorylation of myofilament proteins in response to PKC activation in adult cardiac myocytes. This goal is accomplished by studying myofilament protein phosphorylation in genetically modified myocytes expressing contractile proteins with and without modified phosphorylation sites.
高谈阔论
发表于 2025-3-22 02:03:41
vitro differentiation into cardiomyocytes for transplantation, or as direct sources for cardiac cellular transplantation. In order for cells to be useful for transplantation into injured myocardium, they must be able to survive in the recipient tissue, have limited capacity for replication, and bec
描述
发表于 2025-3-22 06:50:10
ategy for rescue of the various murine cardiac disease models. Finally, gene transfer to the myocardium may also allow genetic targeting of the heart prenatally and postnatally by virus-mediated delivery of Cre recombinase to mice with floxed alleles (.).
小木槌
发表于 2025-3-22 09:32:57
emodelling. .Edward Carmeliet and Johan Vereecke, Katholieke University Leuven,Belgium, have collaborated for over 30 years in cardiacelectrophysiology research. Their studies include the genesis of thenormal action potential, its changes in ischaemia, the effect ofdrugs, and the mechanism of arrhyt
ALIEN
发表于 2025-3-22 14:01:24
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积习已深
发表于 2025-3-22 19:57:45
nhibitors (ACEI), and anti-arrhythmics are also important considerations. Most prospective clinical trials evaluating the efficacy and safety of inhaled bronchodilators have not found a greater risk of cardiac events, however, patients with CVD are usually excluded. Conversely, retrospective studies
ARENA
发表于 2025-3-22 21:50:09
nhibitors (ACEI), and anti-arrhythmics are also important considerations. Most prospective clinical trials evaluating the efficacy and safety of inhaled bronchodilators have not found a greater risk of cardiac events, however, patients with CVD are usually excluded. Conversely, retrospective studies
CALL
发表于 2025-3-23 03:33:08
other proteins have been proposed to contribute. For instance, tropomyosin plays a vital role in stabilizing the thin filament. In addition, cyclase-associated protein 2 (CAP2) has been proposed to regulate thin filament length by severing filamentous actin and sequestering globular actin. Actin-mon
FOVEA
发表于 2025-3-23 08:10:35
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