骇人 发表于 2025-3-25 03:51:16
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Ronald Giemulla,Sebastian Schulz-Stübnermake these cells particularly receptive to changes in mechanical stimuli due to alterations in both blood perfusion and contraction. Recent studies suggest that pericytes, a population of vascular stromal cells supporting capillaries and venules, are highly responsive to conditions of use and disuseGorilla 发表于 2025-3-26 01:27:01
Ronald Giemulla,Sebastian Schulz-Stübneres may also be involved in the pathogenesis of heart failure by converting to myofibroblasts, by secreting fibrogenic and pro-inflammatory mediators, and my regulating microvascular blood flow. Unfortunately, limited information is currently available on the role of pericytes in myocardial diseases.Gullible 发表于 2025-3-26 07:08:09
Ronald Giemulla,Sebastian Schulz-Stübners have been discovered. It has been demonstrated that pericytes from different tissues differ in their properties as well as functions. Even more, pericytes are heterogeneous also within the same organ. This book is will describe the major contributions of pericytes to different organs biology in phGRIPE 发表于 2025-3-26 11:49:38
Ronald Giemulla,Sebastian Schulz-Stübners have been discovered. It has been demonstrated that pericytes from different tissues differ in their properties as well as functions. Even more, pericytes are heterogeneous also within the same organ. This book is will describe the major contributions of pericytes to different organs biology in phMortar 发表于 2025-3-26 13:01:52
Ronald Giemulla,Sebastian Schulz-Stübneres may also be involved in the pathogenesis of heart failure by converting to myofibroblasts, by secreting fibrogenic and pro-inflammatory mediators, and my regulating microvascular blood flow. Unfortunately, limited information is currently available on the role of pericytes in myocardial diseases.colony 发表于 2025-3-26 17:45:45
Ronald Giemulla,Sebastian Schulz-Stübner characterizing changes in single polypeptides by means of O’Farrell’s two-dimensional PAGE (E. and D., 1979; McF. .., 1980). In the case of the orthopoxviruses, studies to date reveal that spontaneous mutations can frequently arise in the form of deletions which may be relatively minor, involving f