Visual-Acuity
发表于 2025-3-26 21:45:27
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esculent
发表于 2025-3-27 03:48:00
r platelet accumulation on the layer of adhered platelets at the injured vessel. Both quantitative and qualitative abnormalities in these platelet glycoproteins can be a cause of platelet dysfunctions and bleeding disorders. In addition, platelet glycoproteins are also important in the pathogenesis
byline
发表于 2025-3-27 05:28:46
Vivian P. Y. Leen the blood group Kell and TPO autoantibody. Kell has been reported to be linked to the locus governing the ability to taste phenylthiocarbamide (PTC) which in turn may be associated with certain forms of thyroid disease . In order to explore further the genetic basis of autoimmune thyroid di
jumble
发表于 2025-3-27 12:33:34
Vivian P. Y. Leen the blood group Kell and TPO autoantibody. Kell has been reported to be linked to the locus governing the ability to taste phenylthiocarbamide (PTC) which in turn may be associated with certain forms of thyroid disease . In order to explore further the genetic basis of autoimmune thyroid di
粗野
发表于 2025-3-27 17:20:15
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NADIR
发表于 2025-3-27 20:53:48
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裙带关系
发表于 2025-3-28 01:45:44
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Alcove
发表于 2025-3-28 04:51:53
Vivian P. Y. Leeression of numerous signaling molecules in SLE T cells. The contribution of each of these mechanisms in the abnormal expression of signaling molecules in SLE T cells is not known. In addition to abnormalities in gene expression, multiple factors, including altered cellular distribution of the protei
Altitude
发表于 2025-3-28 10:02:04
Vivian P. Y. Lees actively using the models presented. Each chapter contains an introductory section that discusses the relevance of the model for a particular disease and for autoimmunity in general. Part I contains methods and protocols to assess immunological and biochemical pathways relevant for disease pathoge
Tractable
发表于 2025-3-28 13:29:23
Vivian P. Y. Leet (FCA)is injected subcutaneously into mice. The immune response, the histological changes, and the genetic susceptibility seen in EAM are similar to those of CB3-induced myocarditis. The third model is experimental autoimmune thyroiditis (EAT). EAT can be induced in genetically susceptible strains