骗子 发表于 2025-3-26 22:50:23
http://reply.papertrans.cn/43/4257/425685/425685_31.pngProjection 发表于 2025-3-27 04:25:15
Lysosomal Degradation of Heparin and Heparan Sulphateroteoglycan (HSPG) is internalised from the cell surface and catabolised with a half-time of 4 to 6 h in rat ovarian granulosa cells (Yanagishita and Hascall, 1984), and by more than 28 h in human colon carcinoma cells (Iozzo, 1987). Studies of both cell types have shown the existence of preliminaryarmistice 发表于 2025-3-27 05:28:13
Heparin Binding Properties of the Carboxyl Terminal Domain of Antistasin 93–119 . (1). This protein contains a specific consensus sequence for heparin binding at its carboxyl terminal end and a region between residues 32 and 48 putatively involved in glycosaminoglycan interactions. The cyclic peptide antistasin 37–48 . and the carboxyl terminal fragment antistasin 93–119尖 发表于 2025-3-27 10:21:58
http://reply.papertrans.cn/43/4257/425685/425685_34.png寡头政治 发表于 2025-3-27 13:41:07
Role of Protein Conformational Changes, Surface Approximation and Protein Cofactors in Heparin-Accelating the activity of these proteinases . is indicated from the well-established link between inherited or acquired deficiencies of antithrombin and the tendency to develop thrombotic disease. Antithrombin is a member of the serpin superfamily of protein proteinase inhibitors and its main target enzllibretto 发表于 2025-3-27 18:17:35
The Interaction of Glycosaminoglycans with Heparin Cofactor II: Structure and Activity of a High-Affmily that are present in plasma at micromolar concentrations. ATIII inhibits several of the proteases involved in coagulation, particularly thrombin and factor Xa, whereas HCII specifically inhibits thrombin.. Both dermatan sulfate and heparin increase the rate of inhibition of thrombin by HCII more俗艳 发表于 2025-3-28 01:58:04
http://reply.papertrans.cn/43/4257/425685/425685_37.png种植,培养 发表于 2025-3-28 02:28:54
The Interaction Between LACI and Heparin fashion, inhibits the factor VIIa/Tissue Factor (TF) catalytic complex (1). Its structure is unique: An acidic amino-terminal domain is followed by three tandem Kunitz-type protease inhibitor domains and a basic carboxyl-terminal region (Fig. 1) (2). The Kunitz-2 domain in LACI is needed for inhibiHUMID 发表于 2025-3-28 09:35:46
http://reply.papertrans.cn/43/4257/425685/425685_39.pngHEDGE 发表于 2025-3-28 12:18:46
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