起来了 发表于 2025-3-26 21:29:51

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nascent 发表于 2025-3-27 04:35:15

Linda C. Burklyd to find, under wide diversities and sharp disagreements, a well-marked current moving in a single direction. Apart from members of traditional schools, the living thinkers of Western Europe are basically concerned with the human life-world, or ., as Husserl called it in his last published work, ..

是他笨 发表于 2025-3-27 08:28:18

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协定 发表于 2025-3-27 11:53:59

Peter J. Larson M.D.,Katherine A. High M.D. weighted generalization have also been found. There are already some expository articles on the classical results as well as on this particular generalization; here we consider the case with a special weight, namely ., and dwell mainly on some recent developments not covered in the earlier expository articles.

reject 发表于 2025-3-27 15:11:55

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截断 发表于 2025-3-27 21:40:03

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Albumin 发表于 2025-3-27 23:54:57

Mechanisms of B-cell Tolerancen division cycles occur between these steps. Surface expression of IgM pins out the immature B cells, which then progressively mature with expression of surface IgD and loss of intracytoplasmic heavy chains.

修饰 发表于 2025-3-28 06:07:31

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欺骗世家 发表于 2025-3-28 09:32:53

Factor Viii Inhibitorskemia. Interestingly, autoantibody patients tend to present with more severe bleeding than hemophilia A patients with a . inhibitor. In both settings, fVIII inhibitors are polyclonal IgG populations directed against multiple epitopes.

松软 发表于 2025-3-28 10:51:00

CD4+ T Cells Specific for Factor VIII as a Target for Specific Suppression of Inhibitor Productionof patients with hemophilia A . Individuals without congenital fVIII deficiency may also develop pathogenic autoimmune inhibitory Ab to fVIII that cause acquired hemophilia, a rare yet severe bleeding disorder (. ). FVIII inhibitors are high affinity IgG, and their synthesis requires the intervention of fVIII specific CD4+ T helper cells.
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查看完整版本: Titlebook: Hemophilia Care in the New Millennium; Dougald M. Monroe,Ulla Hedner,Gilbert C. White Book 2001 The Editor(s) (if applicable) and The Auth