MAOIS
发表于 2025-3-27 00:42:07
Regulation of Insulin Secretion in Vivo,es. Normal glucose regulation is dependent on a closed feedback loop relationship among the liver, peripheral tissues (primarily muscle) and pancreatic islet cells (.). In individuals with normal islet function, the pancreatic islet B-cell adapts to reductions in insulin sensitivity in the hepatic a
笨重
发表于 2025-3-27 04:19:16
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注意力集中
发表于 2025-3-27 07:51:12
Mouse Models of Type 2 Diabetes, recognized to precede beta cell dysfunction, a decreased capacity of the beta cell to compensate for increased insulin production can be demonstrated in healthy first degree relatives of diabetic patients, consistent with a genetic etiology of both defects. Both traits are inherited in a non-Mendel
翻布寻找
发表于 2025-3-27 11:00:30
Candidate Genes for Type 2 Diabetes,0-cell dysfunction that is manifest as inadequate insulin secretion in the face of insulin resistance and hyperglycemia (see Chapters 7 and 8) (.,.). Previous studies have established a clear genetic predisposition for type 2 diabetes (.–.). The concordance rate among monozygotic twins is 50 to 90%
收养
发表于 2025-3-27 14:44:32
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Offensive
发表于 2025-3-27 19:46:19
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流动性
发表于 2025-3-27 22:09:09
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小故事
发表于 2025-3-28 04:11:02
Chronische Arthritis durch Trauma?,lex diseases are characterized as those traits that run in families and are commonly found in the population, but do not exhibit a simple mode of inheritance. They are often referred to as multifactorial because evidence suggests that complex diseases are caused by more than one gene, likely in comb
非秘密
发表于 2025-3-28 09:38:48
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nugatory
发表于 2025-3-28 14:08:00
https://doi.org/10.1007/978-3-662-37022-3ce considered a geneticist’s nightmare (.), diabetes mellitus has recently emerged as a prototype for genetic studies of complex diseases. The ultimate goal of genetic studies is to define etiological subtypes by resolving genetic heterogeneity. The transition of diabetes from a geneticist’s nightma