offense
发表于 2025-3-23 11:05:07
Immunologically Mediated Renal Disease,. However, due to severe side effects with metallodrugs and acquired drug resistance, more metallodrugs are being developed, with improved pharmacological profiles and less side effects. With the rapid development of metallomic strategies in understanding metals in complex biological systems, their
Colonnade
发表于 2025-3-23 14:19:51
https://doi.org/10.1007/978-3-319-55448-8Cancer; Carcinogen; Metal; Molecular medicine; Treatment
密码
发表于 2025-3-23 18:52:11
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cylinder
发表于 2025-3-24 01:15:51
Molecular and Integrative Toxicologyhttp://image.papertrans.cn/e/image/315572.jpg
Nerve-Block
发表于 2025-3-24 04:44:48
2168-4219 related cancers with synthesis and analyses of the available.This book aims to present current state of understanding of the role of metals in human health and disease. As it will be difficult to cover all of the metals, about two scores of them, the authors will instead provide a detailed analysis
negligence
发表于 2025-3-24 10:19:09
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HEAVY
发表于 2025-3-24 14:08:34
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正式演说
发表于 2025-3-24 17:24:00
SpringerBriefs in Speech Technologynogenesis. Limited animal models for arsenic carcinogenesis and limited studies conducted in human cancers caused by arsenic exposure limit the ability to elucidate mechanisms. Research focused on tumors from people suffering from arsenicosis is needed for a clearer understanding of molecular events underlying arsenic-induced carcinogenesis.
火光在摇曳
发表于 2025-3-24 19:24:31
Conducting Polymers and Their Composites, a brief discussion on general molecular pathways of cadmium-induced carcinogenesis. Finally, conclusions are drawn on the existing database in order to identify common and unique molecular pathways of these cancers and to infer biological plausibility.
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发表于 2025-3-24 23:59:26
https://doi.org/10.1007/978-1-349-23809-5ing mechanism of Cr(VI)-induced carcinogenesis. However, recent studies suggest epigenetic modifications also play a crucial role in its carcinogenic mechanism. Therefore, we propose a mechanism of Cr(VI)-induced carcinogenesis that involves both genomic instability and epigenetic modification.