ITCH
发表于 2025-3-26 23:03:50
Epigenetic Alterations to , and , and the Developmental Origins of Mental Disease Riskpring over the life course and contribute to the intergenerational transmission of disease risk. Many psychiatric conditions are now thought to originate in part .. As such, a myriad of gestational exposures may contribute to the biological embedding of poor mental health for the next generation. In
AWL
发表于 2025-3-27 02:16:07
Intergenerational Effects of PTSD on Offspring Glucocorticoid Receptor Methylation parents confers vulnerability to PTSD in offspring. This is underlined by the observation that offspring born to individuals with PTSD exhibit low baseline cortisol levels, a neuroendocrine risk factor for PTSD. Several mechanisms have been proposed to mediate intergenerational inheritance of PTSD,
镇痛剂
发表于 2025-3-27 06:22:40
Epigenetic Biomarkers for Early-Life Adversityl periods in childhood is associated with an increased risk for psychopathology in adulthood, such as mood disorders and mental illness. We have only recently started to uncover some of the mechanisms that may contribute to behavioral changes induced by early-life adversity. Epigenetics modification
抗体
发表于 2025-3-27 13:22:01
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延期
发表于 2025-3-27 13:38:06
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衍生
发表于 2025-3-27 21:48:31
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Seminar
发表于 2025-3-27 23:11:03
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humectant
发表于 2025-3-28 05:37:12
https://doi.org/10.1007/978-3-319-29901-3DNA methylation; epigenetic biomarkers; gene-environmental interactions; neurendocrine differentiation;
Cpap155
发表于 2025-3-28 07:50:22
A Marxist Theory of International Relations, glucocorticoid receptor (GR) takes the central stage in balancing the activity of the HPA axis through its negative feedback on the peptides corticotrophin-releasing hormone and adrenocorticotropic hormone. The activity of GR, in turn, is also controlled by several cofactors including FK506-binding
subacute
发表于 2025-3-28 11:31:36
American Hegemony after the Great Recessionncluding unipolar and bipolar depression and posttraumatic stress disorder (PTSD). Consequently, irregularities in the mRNA expression or protein levels of neuropeptide hormones and receptors involved in related stress pathways can trigger these neurological disorders. As a critical modulator of the