LUT 发表于 2025-3-28 15:17:49
V. N. Chukanov,B. A. Korobitsintery disease (CAD), as emerged only recently. The mechanisms through which epicardial fat can cause atherosclerosis are complex and multifactorial. Its anatomical proximity to the heart, the unique transcriptome, and intense proteasome are the major atherogenic factors of the epicardial adipose tiss针叶 发表于 2025-3-28 22:49:02
http://reply.papertrans.cn/32/3132/313141/313141_42.pngsurmount 发表于 2025-3-29 01:06:49
http://reply.papertrans.cn/32/3132/313141/313141_43.png真实的人 发表于 2025-3-29 04:26:26
Budgetary Constraints on Doctrinal Concepts,le, although results are not univocal. Experimental studies suggest a potential protective role of the brown fat features of EAT against the systolic heart failure. A function of EAT p53 and adiponectin has been also suggested in patients with heart failure. A neuromodulatory role of EAT in heart fa高度表 发表于 2025-3-29 09:45:58
http://reply.papertrans.cn/32/3132/313141/313141_45.pnginterlude 发表于 2025-3-29 11:29:01
,Test and Assessment of Air Purifier ,iver infiltration and insulin resistance. EAT has shown to correlate and predict the risk of developing the metabolic syndrome, although there is no consensus on the EAT threshold risk values, yet. Higher EAT is associated with higher cardiometabolic risk, independently of traditional risk markers.Oligarchy 发表于 2025-3-29 17:51:40
https://doi.org/10.1007/978-3-0348-7970-5aldosterone can lead to accumulation of epicardial adipose tissue (EAT), interfering with functional state. In fact, increased mineralocorticoid signaling is critically involved in the secretion of proinflammatory cytokines by EAT with consequent profibrotic cascade activation in the myocardium. InSPER 发表于 2025-3-29 19:47:36
http://reply.papertrans.cn/32/3132/313141/313141_48.png敏捷 发表于 2025-3-30 03:39:06
http://reply.papertrans.cn/32/3132/313141/313141_49.png险代理人 发表于 2025-3-30 06:36:26
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