分解
发表于 2025-3-28 17:28:10
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烦躁的女人
发表于 2025-3-28 20:49:53
Role of Stem Cells in the Pathogenesis of Endometriosisow indicate that multiple genetic and epigenetic changes (reminiscent of neoplastic processes) are involved in the pathophysiology of endometriosis. Given the molecular similarities between endometriosis and cancer, it is reasonable to apply the “cancer stem cell model” concept to the pathogenesis o
certain
发表于 2025-3-29 01:25:50
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有恶臭
发表于 2025-3-29 03:38:15
Macrophages in Pathophysiology of Endometriosis endometriosis is associated with changes in population and functions of various leukocytes, including macrophages. Macrophages are the most abundant cells found in the peritoneal fluid and are the consistent feature of endometriotic lesion. They infiltrate endometriotic lesions where they undergo a
刺耳的声音
发表于 2025-3-29 08:04:21
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Campaign
发表于 2025-3-29 14:48:49
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NOTCH
发表于 2025-3-29 19:00:06
Roles of Prostaglandin E2 in Endometriosispresence of endometrial-like glands and stroma outside the uterus, commonly on the pelvic peritoneum and ovaries. The major symptoms of endometriosis include pelvic pain, dysmenorrhea, dyspareunia, and infertility. During the past decade, intensive investigations on molecular mechanisms responsible
obviate
发表于 2025-3-29 21:34:38
Sex Steroids and Endometriosis Estrogen metabolism differs considerably in women with a normal endometrium compared to those with estrogen-dependent uterine diseases, including endometriosis, adenomyosis, or fibromas. Altered expression patterns of estrogen receptor (ER)-α/ER-β, progesterone receptor (PR)-A/PR-B, and 17β-hydroxy
负担
发表于 2025-3-30 01:23:52
Aromatase Expression in Endometriosis and Its Significanceon of an endometriotic lesion depends on a sex steroid, estrogen. Aromatase, a key enzyme in estrogen biosynthesis, is highly expressed in the endometriotic tissue, resulting in in situ production of estrogen that, in addition to endocrine estrogen from the ovary, may contribute to the etiology and
同谋
发表于 2025-3-30 07:40:01
Apoptosis in Endometriosistrium from women with endometriosis has some fundamental differences compared with the normal endometrium of women without endometriosis. The differences could contribute to the survival of regurgitating endometrial cells into the peritoneal cavity and the development of endometriosis. One mechanism