chronicle
发表于 2025-3-28 15:19:19
are addressed...Diabetic Nephropathy. will be of interest for nephrologists, diabetologists, internists, transplant physicians, public health professionals, basic scientists, geneticists, epidemiologists, pathologists, and molecular and cell biologists working in the field of diabetes and its complications..978-3-030-06678-9978-3-319-93521-8
expeditious
发表于 2025-3-28 19:58:49
http://reply.papertrans.cn/28/2705/270476/270476_42.png
Armada
发表于 2025-3-28 23:18:25
https://doi.org/10.1007/978-3-531-18970-3uminuric DN phenotype; African-Americans and South Asians are also more likely to progress rapidly to more advanced stages of CKD. Factors such as genetic predisposition, differing access to standard DM care, as well as suboptimal DM-related health-care spending are identified drivers for observed d
杂色
发表于 2025-3-29 05:00:37
http://reply.papertrans.cn/28/2705/270476/270476_44.png
frenzy
发表于 2025-3-29 08:43:43
Handbuch Pädagogische AnthropologieThen, we move to the natural history of DKD progression and relationships between structural changes, renal dysfunction, and diabetes duration. Finally, reversal of DKD lesions following long-term normoglycemia after pancreas transplantation and animal models of DKD are discussed.
上下倒置
发表于 2025-3-29 14:58:37
http://reply.papertrans.cn/28/2705/270476/270476_46.png
famine
发表于 2025-3-29 16:14:19
http://reply.papertrans.cn/28/2705/270476/270476_47.png
洁净
发表于 2025-3-29 22:32:52
http://reply.papertrans.cn/28/2705/270476/270476_48.png
figment
发表于 2025-3-30 00:08:27
Jürgen Straub,Pradeep Chakkarathgulation is believed to disrupt the metabolism of NAD., diminishing the replenishment of the glomerulus with nicotinamide mononucleotide (NMN) from the proximal tubule; this results in Sirt1 downregulation and tight junction protein claudin-1 upregulation in glomerular podocytes. Intracellular nicot
无聊点好
发表于 2025-3-30 06:07:17
Methoden der Film- und Fernsehanalysecollectively play a role in the development and progression of interstitial fibrosis. A comprehensive understanding of the underlying mechanisms and downstream pathways involved in the tubulointerstitial fibrosis is mandatory to develop groundbreaking therapeutic options and novel clinical strategie