太平间
发表于 2025-3-21 16:13:00
书目名称DNA Repair and Human Disease影响因子(影响力)<br> http://figure.impactfactor.cn/if/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease影响因子(影响力)学科排名<br> http://figure.impactfactor.cn/ifr/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease网络公开度<br> http://figure.impactfactor.cn/at/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease网络公开度学科排名<br> http://figure.impactfactor.cn/atr/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease被引频次<br> http://figure.impactfactor.cn/tc/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease被引频次学科排名<br> http://figure.impactfactor.cn/tcr/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease年度引用<br> http://figure.impactfactor.cn/ii/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease年度引用学科排名<br> http://figure.impactfactor.cn/iir/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease读者反馈<br> http://figure.impactfactor.cn/5y/?ISSN=BK0260183<br><br> <br><br>书目名称DNA Repair and Human Disease读者反馈学科排名<br> http://figure.impactfactor.cn/5yr/?ISSN=BK0260183<br><br> <br><br>
patriot
发表于 2025-3-21 21:42:15
https://doi.org/10.1007/978-3-030-80606-4oth transcription and NER. . and . encode two subunits of TFIIH, and also TTDA has been recendy identified as a new component of the TFIIH complex. The discovery of this unexpected link between DNA repair and transcription was crucial to rationalize the TTD pathological phenotype as well as the puzz
牛马之尿
发表于 2025-3-22 01:57:58
https://doi.org/10.1007/978-3-642-31322-6BRCA1 in discrete nuclear .. FANCD2 is also independently phosphorylated by ATM in response to ionising radiation and interacts with the MRE1 l/Rad50/NBSl complex, which is directly involved in homologous recombination DNA repair pathway and in cell cycle checkpoint response to DNA damage. Available
Nostalgia
发表于 2025-3-22 05:36:34
https://doi.org/10.1007/978-3-642-31322-6eview is focused on the potential functions of . in both DNA repair and cell cycle checkpoint regulation and how deficiencies in these overlapping functions can lead to some of the phenotypic features of AT patients.
aggravate
发表于 2025-3-22 12:43:48
https://doi.org/10.1007/978-3-030-37293-4cells has identified the involvement of potential DNA repair factors in such pathways. Studies by us and others have shown the association between the DNA repair factors and telomere function in mammalian cells. Mice deficient in a DNA-break sensing molecule, PARP-1 (poly -ribopolymerase), have
Radiculopathy
发表于 2025-3-22 14:04:36
Human Premature Aging Disorders and Dysfunction of DNA Repair,ocalization studies all suggest WRN’s participation in multiple DNA metabolic pathways for maintaining genomic integrity. CS arises from mutations in the CSA and CSB genes. While CSA belongs to “WD” repeat containing protein family, CSB is a member of SNF2-like family with only a DNA stimulated ATPa
Radiculopathy
发表于 2025-3-22 19:14:51
Trichothiodystrophy,oth transcription and NER. . and . encode two subunits of TFIIH, and also TTDA has been recendy identified as a new component of the TFIIH complex. The discovery of this unexpected link between DNA repair and transcription was crucial to rationalize the TTD pathological phenotype as well as the puzz
有法律效应
发表于 2025-3-22 23:05:54
The Fanconi Anemia/BRCA Pathway,BRCA1 in discrete nuclear .. FANCD2 is also independently phosphorylated by ATM in response to ionising radiation and interacts with the MRE1 l/Rad50/NBSl complex, which is directly involved in homologous recombination DNA repair pathway and in cell cycle checkpoint response to DNA damage. Available
cardiovascular
发表于 2025-3-23 04:53:39
Is Ataxia Telangiectasia a Result of Impaired Coordination between DNA Repair and Cell Cycle Checkpeview is focused on the potential functions of . in both DNA repair and cell cycle checkpoint regulation and how deficiencies in these overlapping functions can lead to some of the phenotypic features of AT patients.
档案
发表于 2025-3-23 08:54:40
Orchestration of Telomeres and DNA Repair Factors in Mammalian Cells,cells has identified the involvement of potential DNA repair factors in such pathways. Studies by us and others have shown the association between the DNA repair factors and telomere function in mammalian cells. Mice deficient in a DNA-break sensing molecule, PARP-1 (poly -ribopolymerase), have