BRAND
发表于 2025-3-30 11:59:14
A Yeast Expression System of High Efficiency for Producing Recombinant Enzymes,entified and they are the . ADP-ribosyl cyclase and the mammalian antigens, CD38 and CD157 (formerly called BST-1) . The sequence homology among these three enzymes is shown in Figure 1. All three enzymes are widespread in occurrence and are localized in a variety of tissues . The novel
委托
发表于 2025-3-30 15:16:19
Novel Cycling Assays for cADPR and NAADP,pecific and sensitive assays for cADPR and NAADP be widely available for monitoring their endogenous levels under a variety of physiological conditions. The first assay for cADPR was a bioassay based on its Ca. releasing activity in sea urchin egg homogenates . Using this assay, it was demonst
兽群
发表于 2025-3-30 17:02:27
Pharmacology of Cyclic ADP-Ribose and NAADP,that inositol trisphosphate mobilizes Ca. stores in the endoplasmic reticulum . Also present in the organelle is another Ca. release channel, the ryanodine receptor. The discovery of two other Ca. signaling molecules, cyclic ADP-ribose (cADPR) and nicotinic acid adenine dinucleotide phosphate (NA
埋葬
发表于 2025-3-30 22:53:20
Separate but Interacting Calcium Stores,nd internal. One of the most dramatic examples occurs during fertilization. Immediately after sperm-egg fusion, a highly localized Ca. elevation is initiated right at the fusion site. This spark of Ca. then grows into a wave propagating across the entire egg. Intriguingly, the initiation of apoptoti
节约
发表于 2025-3-31 04:14:47
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Seminar
发表于 2025-3-31 06:10:13
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连接
发表于 2025-3-31 12:35:55
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过滤
发表于 2025-3-31 13:27:23
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Infect
发表于 2025-3-31 20:40:34
,The CD38-Cyclic ADP-Ribose Signal System in Pancreatic ß-Cells,of insulin, and proposed a unifying model for the action of the diabetogenic agents alloxan and streptozotocin on pancreatic β-cells (Figure 1) . Central to the model are breaks in the nuclear DNA of β-cells, resulting from either an accumulation of free radicals or from the alkylation of DNA.
改正
发表于 2025-3-31 22:22:19
The Role of cADPR and NAADP in T Cell Calcium Signaling and Activation,years ago many researchers believed that Ca. release in T cells is exclusively operated by D-.-inositol 1,4,5-trisphosphate (InsP3; reviewed in ref. 4) while the capacitative mechanism of Ca. entry proposed by Putney was thought to play a central role in the influx process.