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978-1-4939-4704-1Springer Science+Business Media New York 2015cyanosis 发表于 2025-3-22 02:35:08
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Clinical Hypertension and Vascular Diseaseshttp://image.papertrans.cn/c/image/226386.jpgEviction 发表于 2025-3-22 12:05:36
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https://doi.org/10.1007/978-3-319-21103-9e is steadily escalating to hazardous proportions due to the increasing incidence of hypertension, diabetes, metabolic syndrome, and other risk factors. Hypertension can be a cause or consequence of CKD. Nevertheless, the coexistence of hypertension and CKD portends poor prognosis due to the occurreExcitotoxin 发表于 2025-3-22 21:17:08
Story Credibility in Narrative Argumentsute to hypertension in these patients, including volume expansion and activation of the renin–angiotensin–aldosterone system. This chapter deals with the role of the sympathetic nervous system in hypertension and CKD progression in these patients and with the mechanisms that can contribute to the acnitroglycerin 发表于 2025-3-23 02:37:59
https://doi.org/10.1007/978-1-349-06892-0 models and new options to block the renin–angiotensin–aldosterone axis such as renin inhibitors, angiotensin II type 2 receptor agonists, angiotensin vaccines and aldosterone synthase inhibitors are under scrutiny with variable results so far. Interventions in different hormones and autacoids, enzy凝乳 发表于 2025-3-23 08:46:30
Key Currencies and Financial Centresor blocker (ARB) or a direct renin inhibitor (DRI) similarly reduces blood pressure (BP) when each is used as monotherapy in patients with hypertension. Both ACE inhibitors and ARBs also slow down the progressive decline in renal function, which marks renal injury, particularly in patients with diab