consent
发表于 2025-3-25 05:09:29
https://doi.org/10.1007/978-3-540-76890-6d cell was less able to buffer the changes in the intracellular calcium, thus providing a biological basis for the arrhythmogenicity of the hypertrophied heart. These various modifications may provide a new key for future pharmaceutical research.
BOOST
发表于 2025-3-25 10:09:21
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蛤肉
发表于 2025-3-25 14:23:10
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时代错误
发表于 2025-3-25 16:56:20
Cardiovascular cyclic nucleotide phosphodiesterases and their role in regulating cardiovascular func diseases. More potent selective inhibitors of the cGMP-PDE isozyme are needed to determine whether these pharmacological potentiators of EDRF and ANP will be useful in the therapy of angina, hypertension or heart failure.
LIMN
发表于 2025-3-25 21:51:28
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圣歌
发表于 2025-3-26 00:55:04
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Aspirin
发表于 2025-3-26 04:18:21
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使习惯于
发表于 2025-3-26 09:37:43
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Mri485
发表于 2025-3-26 16:00:57
Eine allgemeine Konvergenztheorie,n function may be the basis for .-receptor uncoupling in IDC and ISCDC, whereas in PPH, this phenomenon may result from altered adenylyl cyclase function. Catalytic subunit activity of adenylyl cyclase is decreased in order of increasing pulmonary hypertension in right-ventricular preparations from
HALO
发表于 2025-3-26 18:30:27
https://doi.org/10.1007/978-3-540-76790-9dilated cardiomyopathy and 48% in ischemic cardiomyopathy, although pertussis toxin substrates were only increased by 40% in dilated cardiomyopathy and no change was observed in ischemic cardiomyopathy. In cardiomyopathy tissue, an inverse relationship was observed between the increase of G. and the