切割
发表于 2025-3-28 15:50:23
Cell-cell and cell-matrix adhesion molecules in human heart and lung transplantsptor-ligand molecules. The molecules mediating intercellular adhesion and activation are classified as integrin, immunoglobulin and selectin families. In the present study the patterns of their cellular expression in human heart and lung transplants are described in normal state and during transplan
星星
发表于 2025-3-28 20:00:31
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STAT
发表于 2025-3-29 01:39:39
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Esalate
发表于 2025-3-29 07:08:28
Differences between atrial and ventricular protein profiling in children with congenital heart diseamples were obtained during surgery from patients with normoxemic (ventricular and atrial septal defects) and hypoxemic (tetralogy of Fallot) diseases. Protein fractions were isolated by stepwise extraction from both right ventricular and atrial musculature. The concentration of total atrial protein
表示问
发表于 2025-3-29 08:26:24
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曲解
发表于 2025-3-29 13:49:02
Factors involved in capillary growth in the heart and changes in cardiac metabolism or heart rate elicited by modification of thyroid hormone levels. Capillary growth in all these conditions can be linked with increased coronary blood flow, decreased heart rate, or both. This paper brings evidence that, although increased blood flow due to long-te
Cougar
发表于 2025-3-29 19:25:36
Biomechanical signals in the coronary artery triggering the metabolic processes during cardiac overlthe human coronary artery in which the remarkable wall thickness is due namely by the intima thickness. Deformation in length and diameter of conduit coronary arteries, due to the left and right ventricle volume increase, has been defined in non-beating canine heart. Ramus interventricularis anterio
hair-bulb
发表于 2025-3-29 22:39:16
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臆断
发表于 2025-3-30 02:43:17
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无脊椎
发表于 2025-3-30 05:46:41
Angiotensin converting enzyme inhibitors, left ventricular hypertrophy and fibrosisions, which are not related to hemodynamic changes and which cannot be explained only by interference with the renin angiotensin system (RAS) by means of an inhibition of angiotensin II (ANG II) formation. Since ACE is identical to kininase II, which inactivates the nonapeptide bradykinin (BK) and r