憎恶
发表于 2025-3-25 03:32:58
https://doi.org/10.1007/978-3-531-90185-5cytes this phenomenon has not been confirmed. The purpose of the present study was to confirm the effect of (1, 4, 5)-IP. on Ca. release from SR in cardiac myocytes. The effect of IP. on Ca. release from SR in hypertrophic cardiac cells was also determined..We examined the effects of IP. on Ca. rele
Trypsin
发表于 2025-3-25 10:37:32
https://doi.org/10.1007/978-3-531-90185-5le activation with different maneuvers was mapped with digital imaging microscopy. During 50 Hz electrical activation (tetanus), the aequorin light intensity from different locations in the muscle fiber rose with very similar time course. Caffeine (10 mM) application, on the other hand, caused aequo
放肆的我
发表于 2025-3-25 13:11:01
Gert Pickel,Detlef Pollack,Jörg Jacobsrant, and can not provoke any heart-specific disease. Why the proteins can provoke such lethal autoimmune myocarditis has not been completely elucidated. Shortly after cardiac infection or myocardial ischemia, these proteins may work as the antigen for the autoimmune myocardites. First of all, the r
craving
发表于 2025-3-25 16:49:14
Gert Pickel,Detlef Pollack,Jörg Jacobsstigate whether a different origin of the myocardial sample or pretreatment with the cardioprotective agent 2, 3-butanedione-monoxime (BDM) influences the force-frequency- relationship in electrically driven muscle strips taken from failing and nonfailing human myocardium. With as well as without pr
混杂人
发表于 2025-3-25 20:30:28
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Ballad
发表于 2025-3-26 00:11:04
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避开
发表于 2025-3-26 04:23:48
https://doi.org/10.1007/978-3-531-90378-1s of aging on human mitochondrial DNA. 8-Hydroxy-deoxyguanosine, a product of free radical damage to deoxyguanosine, is reported to cause random point mutations. In human mitochondrial DNA, 8-hydroxy-deoxyguanosine increased exponentially with age, and the population of mitochondrial DNA with deleti
印第安人
发表于 2025-3-26 09:33:17
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昏暗
发表于 2025-3-26 15:03:25
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全面
发表于 2025-3-26 16:49:46
https://doi.org/10.1007/978-3-531-90378-1f saturation transfer using .P-NMR during the preischemic and reperfusion periods. The hearts were made ischemic for 15 min, followed by 60 min of reperfusion. PRP was almost completely depressed, and recovered to 60% of the control level (preischemic period) after reperfusion. The ATP level during