烧烤
发表于 2025-3-23 13:18:02
Sex Steroid-Dependent and -Independent Angiogenesis in Uterine Endometrial CancersEGF in well-differentiated uterine endometrial cancers and to bFGF in poorly differentiated uterine endometrial cancers. Therefore, even if dedifferentiation and angiogenic switching occur due to advancement and long-term hormone therapy, the inhibition of ETS-1, along with main angiogenic factors,
ironic
发表于 2025-3-23 15:21:55
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思想灵活
发表于 2025-3-23 21:58:57
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多山
发表于 2025-3-24 00:36:26
Robert A. Jerin,Beverly Dolinsky three cancer cells in the presence of HGF was observed by Boyden’s chamber assay. HGF enhanced the activation of MMP-2 and -9 by zymography of MMPs. HGF strongly induced MMP-9 mRNA expression in stromal cells, but had little effect on MMP-2 mRNA. MT1-MMP mRNA was detected only in KLE and stromal ce
不能约
发表于 2025-3-24 05:49:28
https://doi.org/10.1057/9780230206182 vitro study. Quantitative analysis of proteins was performed by immunoblotting. Expression of cyclin D1, cyclin E, cyclin A, p53, and p27 was positively correlated with Ki-67 expression. Expression of cdk2, cyclin Dl, cyclin E, cyclin A, p53, and p27 was positively associated with histological grad
Neolithic
发表于 2025-3-24 10:12:51
https://doi.org/10.1057/9780230206571(TCDD) induce CYP1A1-dependent activity or reporter gene activity in cells transfected with constructs containing dioxin-responsive elements as promoters. Estrogen responsiveness was also confirmed in these cells, as evidenced by gene/reporter gene assay and the induction of cell proliferation by 17
RENIN
发表于 2025-3-24 14:24:49
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发现
发表于 2025-3-24 18:07:48
Conclusion: The History of Anaesthesia,se-1/2, suggesting that API-59 inhibits the AKT pathway at the AKT kinase level..We next investigated whether API-59 induces apoptosis in these endometrial cancer cell lines. Exposure to API-59 induced significant apoptosis in both Ishikawa and RL95-2 cancer cell lines, which express elevated AKT ki
Evacuate
发表于 2025-3-24 21:06:56
https://doi.org/10.1057/9780230222977ts its cellular responses via two high-affinity tyrosine kinase receptors, namely VEGFR-1 and VEGFR-2. Until recently, it was presumed that the majority of cellular effects induced by VEGF were mediated by VEGFR-2, whereas VEGFR-1 acted as a decoy. However, results from our laboratory and others hav
Hemoptysis
发表于 2025-3-25 01:28:41
https://doi.org/10.1057/9780230222977EGF in well-differentiated uterine endometrial cancers and to bFGF in poorly differentiated uterine endometrial cancers. Therefore, even if dedifferentiation and angiogenic switching occur due to advancement and long-term hormone therapy, the inhibition of ETS-1, along with main angiogenic factors,