Armory 发表于 2025-3-28 14:57:07
http://reply.papertrans.cn/23/2220/221942/221942_41.png气候 发表于 2025-3-28 20:32:58
https://doi.org/10.1007/978-3-319-66114-8Adult congenital heart disease; Cardiac Genomics; Familial cardiomyopathies; Gene sequencing; Inherited植物茂盛 发表于 2025-3-29 00:17:35
978-3-319-88179-9Springer International Publishing AG 2018讲个故事逗他 发表于 2025-3-29 07:09:38
25-1 - 25-95: C7Cl2F4N2 - C7H5ClF3N,fect the individual at any stage from fetal to adult life. It also encompasses abnormalities encountered in postnatal adaptation, such as persistence of the ductus arteriosus. Cardiovascular abnormalities, as a group, are the most common cause of congenital anomaly and account for around 1% of live births.jealousy 发表于 2025-3-29 10:52:40
http://reply.papertrans.cn/23/2220/221942/221942_45.png舔食 发表于 2025-3-29 14:11:34
http://reply.papertrans.cn/23/2220/221942/221942_46.png争议的苹果 发表于 2025-3-29 18:07:52
24-96 - 24-222: C6H8ClN3 - C7ClF5O, therapy, this heterogeneity remains challenging to classify and, ultimately, a combined approach is necessary. As genomic medicine progresses and we enter an era of personalised medicine, our ability to develop an organic classification system able to deal with complexity will be key.–scent 发表于 2025-3-29 21:24:02
24-1 - 24-95: C6H5Cl4N - C6H8ClNO2, help identify other individuals at risk of developing the disease phenotype. The following chapter provides insight into the genetic aetiology underlying these diverse disease processes and the clinical management of these inherited cardiomyopathies.平项山 发表于 2025-3-30 03:58:28
23-116 - 23-221: C6H4Cl2N2O2S - C6H5Cl3Sn, clinical history and pathology investigations. Genetic tests in probands and cascade family screening in clinically phenotyped families provide the basis for segregation studies that are major contributors to the assignment of a causative role of mutations in families.思想流动 发表于 2025-3-30 06:35:22
26-163 - 26-222: C8H4Cl2N2 - C8H6Cl2O,ugh it is often associated with other congenital cardiac anomalies it can also be seen in association with dilated, hypertrophic and restrictive cardiomyopathies and in the absence of any cardiac defects.