PAD416 发表于 2025-3-23 09:52:44

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Mri485 发表于 2025-3-23 16:02:38

Environmental Science and Engineering]. transient (CaT) decides about the strength of contraction. Increases in L-type Ca. current, SERCA activity, SR Ca. load and fractional release, IP. signalling and . all increase CaT amplitude. Under conditions of Ca. overload, SR Ca. release also occurs spontaneously, i.e. in the absence of

前面 发表于 2025-3-23 18:31:24

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AWE 发表于 2025-3-24 00:40:59

https://doi.org/10.1007/978-94-010-0066-6f endogenous mechanisms that can contribute to the regulation of the myocardial contractility and illustrates the various feedback loops and signalling pathways that can be induced via corresponding modulation of the cardiac function. A detailed description of the underlying cellular and molecular m

Neonatal 发表于 2025-3-24 05:14:18

https://doi.org/10.1007/978-3-319-07055-1hormones, cytokines and neurotransmitters have been identified that trigger cardiac hypertrophy and specifically those related to the sympathetic nervous system, and the renin-angiotensin system is of importance for the transition to heart failure. An increase in the translational capacity is the ba

GIBE 发表于 2025-3-24 08:32:32

Martin Adam,Stephan Schäffler,Anna Brauny in particular evolved to the level that they are considered decisive in disease development. Indeed, their manipulation in vitro as well as in vivo influence cardiomyocyte function and cardiac disease outcome. Substrates subject to degradation range from single proteins (ubiquitin-proteasome pathw

bisphosphonate 发表于 2025-3-24 11:50:14

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adipose-tissue 发表于 2025-3-24 18:46:38

Ways to Study the Biology of Cardiomyocytesemodeling. Function of cardiomyocytes is mostly characterized by load-free cell shortening with remarkable reproducible results between cardiomyocytes from different species. Molecular aspects of cardiac hypertrophy can be analyzed by quantification of protein synthesis, protein degradation, and cel

cylinder 发表于 2025-3-24 20:25:30

Excitation–Contraction Coupling of Cardiomyocytes]. transient (CaT) decides about the strength of contraction. Increases in L-type Ca. current, SERCA activity, SR Ca. load and fractional release, IP. signalling and . all increase CaT amplitude. Under conditions of Ca. overload, SR Ca. release also occurs spontaneously, i.e. in the absence of

Parley 发表于 2025-3-25 01:51:03

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查看完整版本: Titlebook: Cardiomyocytes – Active Players in Cardiac Disease; Klaus-Dieter Schlüter Book 2016 Springer International Publishing Switzerland 2016 car