lexicographer 发表于 2025-3-21 18:38:31

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商店街 发表于 2025-3-21 20:34:19

PI3K/AKT Pathway and the Epithelial-Mesenchymal Transitionhibitor .. By some estimates, . carries gain-of-function mutations in 32% of colorectal cancers, 36% of hepatocellular carcinomas, 36% of endometrial carcinomas, 25% of breast carcinomas, 15% of anaplastic oligodendrogliomas, and 5% of medulloblastomas and anaplastic astrocytomas (recently reviewed

linear 发表于 2025-3-22 01:03:35

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osculate 发表于 2025-3-22 07:32:02

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Consensus 发表于 2025-3-22 08:59:33

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闪光你我 发表于 2025-3-22 15:08:19

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闪光你我 发表于 2025-3-22 18:49:46

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ARCHE 发表于 2025-3-23 01:07:17

Myc and Control of Tumor Neovascularization control of a heavy chain gene enhancer (Dalla-Favera et al. 1982; Taub et al. 1982). Additionally, double minute chromosomes (dmin) containing amplified copies of the c-Myc gene are commonly detected in acute myeloid leukemia (AML) and myelodysplastic syndromes (MDS) (Storlazzi et al. 2004). In par

Barrister 发表于 2025-3-23 02:17:50

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xanthelasma 发表于 2025-3-23 05:45:00

Ink4a Locus: Beyond Cell Cycleactivation of p16 include melanoma, adenocarcinoma of the breast, squamous cell carcinomas of the lung, pancreatic adenocarcinomas, and colorectal carcinomas (Baylin et al. 1998; Pollack, Pearson, and Hayward 1996). In pancreatic adenocarcinoma, the frequency of p16 inactivation approaches 100% (Cal
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查看完整版本: Titlebook: Cancer Genome and Tumor Microenvironment; Andrei Thomas-Tikhonenko Book 2010 Springer-Verlag New York 2010 Chromosom.angiogenesis.cell.gen