描绘
发表于 2025-3-23 13:41:41
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Panacea
发表于 2025-3-23 17:37:55
Jennifer Limond,Anna-Lynne Ruth Adlamluorouracil, 5-f luorodeoxyuridine, or 5-fluorouridine (1). Inhibition of thymidylate synthase can lead to a depletion of thymine nucleotides and result in DNA synthesis inhibition due to substrate deficiency.
BARB
发表于 2025-3-23 18:59:23
Stan Smith,Nelson Butters,Eric Granholming, proton (.H) NMR has become widely used for investigations of tissue water and to produce detailed tomographic images which reflect the spatial distribution of tissue water and the distribution of proton relaxation.
POWER
发表于 2025-3-23 22:37:23
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jealousy
发表于 2025-3-24 03:23:50
Evaluation of Neurotoxic Syndromes, patients receiving systemic cancer treatment. In this volume we seek to give an overview of known molecular and clinical factors which may result in cancer drug resistance and the probable interplay of these factors in the evolution of resistance in man. Finally, we discuss treatment approaches tha
exophthalmos
发表于 2025-3-24 07:31:36
David W. Ellis,Anne-Lise Christensennd biological activities, which include cytotoxicity, carcinogenicity, mutagenicity, teratogenicity and use in the chemotherapy of human cancer, have been extensively reviewed (1–7). Most studies of the acute toxicity of the nitrosamines have been carried out with the shorter chain dialkyl and simpl
mortuary
发表于 2025-3-24 12:41:09
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FLOUR
发表于 2025-3-24 18:05:29
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全神贯注于
发表于 2025-3-24 21:14:53
Biochemical Strategy of Cancer Cells and the Targeting of Chemotherapy with Tiazofurin, Acivicin, antions of strategic nucleotides and amino acids are stringently linked with neoplastic transformation and progression. Parameters that are not stringently linked yield no pattern. The conclusion was drawn that what is important about cancer is ordered; what is not, is the random element and the diversity. This field was recently reviewed (2,3).
Cubicle
发表于 2025-3-25 01:13:58
Implications of Therapy-Induced Genetic/Epigenetic Modulations of Tumor Progression and Tumor Cell Hcy (i.e., “cryptic”) mutants which had spontaneously undergone a genetic mutation (making the cells antibiotic-resistant) prior to exposure of the bacteria to the antibiotic; the presence of the drug then acts as a potent selection pressure to promote the overgrowth of the antibiotic-resistant mutants.