Helmet
发表于 2025-3-21 19:19:02
书目名称Brain Injury影响因子(影响力)<br> http://impactfactor.cn/if/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury影响因子(影响力)学科排名<br> http://impactfactor.cn/ifr/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury网络公开度<br> http://impactfactor.cn/at/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury网络公开度学科排名<br> http://impactfactor.cn/atr/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury被引频次<br> http://impactfactor.cn/tc/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury被引频次学科排名<br> http://impactfactor.cn/tcr/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury年度引用<br> http://impactfactor.cn/ii/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury年度引用学科排名<br> http://impactfactor.cn/iir/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury读者反馈<br> http://impactfactor.cn/5y/?ISSN=BK0190203<br><br> <br><br>书目名称Brain Injury读者反馈学科排名<br> http://impactfactor.cn/5yr/?ISSN=BK0190203<br><br> <br><br>
天赋
发表于 2025-3-21 20:14:50
openSAP – der Enterprise-MOOC-Pioniersent novel targets for therapies aimed at limiting damage after acute or chronic CNS injury. This chapter will provide a general review of death receptors and their signaling mechanisms, focusing on the two best characterized death receptors, Fas and TNF-α.
jocular
发表于 2025-3-22 01:57:39
Machen elektromagnetische Felder krank?l disintegration, nuclear condensation, and digestion of DNA. Finally, the engulfment of the dead cell or cell fragments by phagocytosis peacefully terminates the death program. Incomplete execution of programmed cell death may redirect the cell to necrosis, which is an unfavorable event for the org
杠杆支点
发表于 2025-3-22 05:35:45
Machiavelli in Contemporary Media neurocytoskeletal elements sustain damage as a result of trauma, potentially contributing to neuronal dysfunction. Some of the major neurocytoskeletal proteins include neurofilaments (NFs), tubulin, microtubule-associated proteins (MAPs) such as MAP2 and tau, actin, and spectrin. Because cytoskelet
焦虑
发表于 2025-3-22 09:35:45
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偶像
发表于 2025-3-22 13:51:37
The Multifaceted Role of Adenosine in Experimental and Clinical Traumatic Brain Injury,thermia—putative pharmacological agents that might simultaneously or sequentially confer multiple beneficial effects in the injured brain. One endogenous mediator that fits this category is adenosine—which has the potential to favorably influence excitotoxicity, energy failure, hypoperfusion, calciu
instructive
发表于 2025-3-22 18:10:03
Death Receptors in Acute Brain Injury,sent novel targets for therapies aimed at limiting damage after acute or chronic CNS injury. This chapter will provide a general review of death receptors and their signaling mechanisms, focusing on the two best characterized death receptors, Fas and TNF-α.
PANIC
发表于 2025-3-22 22:51:06
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遗传学
发表于 2025-3-23 03:50:13
Neurocytoskeletal Changes Following Traumatic Brain Injury, neurocytoskeletal elements sustain damage as a result of trauma, potentially contributing to neuronal dysfunction. Some of the major neurocytoskeletal proteins include neurofilaments (NFs), tubulin, microtubule-associated proteins (MAPs) such as MAP2 and tau, actin, and spectrin. Because cytoskelet
注意力集中
发表于 2025-3-23 07:10:15
Reproductive Hormones as Neuroprotectants in Brain Injury,d molecular mechanisms by which estrogen salvages brain are detailed. We focus on estrogen’s mechanisms since there is a large, and rapidly developing, understanding of this hormone’s neuroprotective potential. Limited data are available at present for progesterone or testosterone (reviewed in .; .)