压倒性胜利 发表于 2025-3-25 03:40:50
978-3-031-13011-3Springer Nature Switzerland AG 2023OVERT 发表于 2025-3-25 08:23:50
http://reply.papertrans.cn/19/1894/189312/189312_22.pngLumbar-Stenosis 发表于 2025-3-25 15:01:46
http://reply.papertrans.cn/19/1894/189312/189312_23.pngApoptosis 发表于 2025-3-25 18:05:34
Hypertension and Type 2 Diabetesuences. The main driving mechanisms of this association are maladaptive endothelial dysfunction, renin-angiotensin-aldosterone system (RAAS) and sympathetic overactivity, abnormal sodium handling, renal dysfunction, and vascular stiffness. Blood pressure (BP) elevation in T2DM has a characteristic pFRAX-tool 发表于 2025-3-25 23:53:43
http://reply.papertrans.cn/19/1894/189312/189312_25.pngConflagration 发表于 2025-3-26 03:15:10
Office/Out-of-Office Blood Pressure Measurementsion-induced organ damage and the risk of major cardiovascular complications and mortality, emerging from studies conducted in mixed cohorts of diabetic and nondiabetic subjects, has been substantially confirmed in the specific context of diabetes. Consequently, self-measured home BP and 24-h BP moniJogging 发表于 2025-3-26 07:44:22
Laboratory Indices/Bioimaging possibly on an annual basis. In patients with hypertension and concomitant diabetes, the risk of developing cardiovascular disease (CVD), macrovascular diseases, and/or microvascular diseases is two- to fivefold greater than in patients with either hypertension or DM. In patients with DM, the preseRotator-Cuff 发表于 2025-3-26 09:54:02
Molecular Mechanisms Underlying Vascular Disease in Diabetes in diabetes is typically associated with atherosclerosis and calcification leading to heart disease and stroke. Microvascular disease leads to retinopathy, nephropathy and neuropathy. Clinically this manifests as reduced vision and blindness, kidney dysfunction and peripheral neuropathy—major complsebaceous-gland 发表于 2025-3-26 13:21:43
http://reply.papertrans.cn/19/1894/189312/189312_29.pngFUSC 发表于 2025-3-26 17:42:15
Mechanisms of Diabetic Nephropathy in Humans and Experimental Animals to the utility of experimental animal models. Pathologically, DKD is characterized by glomerular basement membrane thickening, mesangial matrix expansion, glomerulosclerosis, and tubulointerstitial fibrosis. These changes correlate clinically with the development of albuminuria and hypertension and