改变立场
发表于 2025-3-23 10:24:53
Moral auf Umwegen: „Das Wort zum Sonntag“al osteoarthritis (Alla-Kokko et al., 1990). The chromosomal locations of genes for numerous other inherited diseases of unknown biochemical etiology have been defined, although the precise genes have not been discovered yet (for review see Human Gene Mapping 10, 1989).
hieroglyphic
发表于 2025-3-23 14:11:13
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Ophthalmoscope
发表于 2025-3-23 18:44:20
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停止偿付
发表于 2025-3-24 02:08:19
Book 1993ion of depressive illness from a variety of perspectives that include those of the patient, the clinician, and the psychiatric researcher. It de scribes the major subtypes of depressive illness and places them in the context of the life cycle. The second volume in this series is entitled Models of
BLA
发表于 2025-3-24 02:26:50
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NOT
发表于 2025-3-24 07:06:20
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Modify
发表于 2025-3-24 11:50:21
Sleep Disorders in Depressive Disorders,r total sleep deprivation, but approximately 60% of depressed patients note transient antidepressant effects after sleep deprivation. These basic observations suggest not only that mood and sleep interact with each other, but that they interact differently in depressed and nondepressed individuals.
罗盘
发表于 2025-3-24 17:45:06
Thyroid Axis and Depressive Disorders,tion. The major focus of this chapter will be on studies examining the interaction between the thyroid axis and depression. However, we start with a brief overview of reasons for interest in a role of the thyroid axis in depression.
愤世嫉俗者
发表于 2025-3-24 20:24:55
Ansatzpunkt 3: Generative Kommunikatione was often accompanied by the development of depressive symptoms. Similarly, the first-generation antidepressants were early reported to enhance biogenic amine activity through inhibition of monoamine oxidase or blockade of synaptic reuptake. Thus the catecholamine theory of depression was first published about 25 years ago.
炼油厂
发表于 2025-3-25 02:10:08
Gaba and Depression,e was often accompanied by the development of depressive symptoms. Similarly, the first-generation antidepressants were early reported to enhance biogenic amine activity through inhibition of monoamine oxidase or blockade of synaptic reuptake. Thus the catecholamine theory of depression was first published about 25 years ago.