变化无常
发表于 2025-3-30 09:41:28
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河流
发表于 2025-3-30 14:40:09
Extracellular matrix degrading metalloproteinases in the pathogenesis of arteriosclerosis,ncentration-dependent manner, Ro 31–4724 also inhibited outgrowth. Rabbit aortic smooth muscle cells were further shown to release MMPs, namely a 95 and a 72 kDa gelatinases that were inhibited by Ro 31–4724 and Ro 31–7467. The evidence suggests that degradation of basement membrane by gelatinase is
奇怪
发表于 2025-3-30 18:38:11
Vascular renin-angiotensin-system, endothelial function and atherosclerosis?,ion of elastic arteries upregulates the local renin-angiotensin system in these arteries and thereby downregulates nitric oxide releasability. Enhanced local synthesis of the trophic factor angiotensin-II and reduced releasability of the antitrophic factor nitric oxide appear to cooperate in the tro
Indent
发表于 2025-3-30 23:29:13
Endothelial dysfunction in atherosclerosis,review, evidence for these alterations will be considered, potential interventions for restoring endothelium-dependent relaxations examined, and the possible impact of endothelial dysfunction in atherosclerosis considered.
Hemoptysis
发表于 2025-3-31 02:44:10
New determinants of the uptake of atherogenic plasma proteins by arteries,ectly supports the evidence that LDL leaves the plasma by transcytosis through the endothelial cells..In anesthetized rabbits and in conscious rats the uptake of LDL, methylated to prevent its removal by high-affinity receptors (m-LDL), is significantly increased by noradrenaline and by adrenaline a
演绎
发表于 2025-3-31 08:15:46
Assessment of endothelial modulation of coronary vasomotor tone: Insights into a fundamental functilopment of atherosclerosis, and impaired endothelial vasodilation is the predominant mechanism underlying inappropriate vasoconstriction in atherosclerosis. However, endothelial vasodilator dysfunction is not only confined to atherosclerotic epicardial vessels, but may also extend into the coronary
议程
发表于 2025-3-31 12:53:13
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男生戴手铐
发表于 2025-3-31 15:38:14
Therapeutic approaches to the control of coronary atherosclerosis,h lower plasma cholesterol provide one approach. Another approach is to use calcium antagonists which not only lower blood pressure, but also directly interfere with some of the metabolic events involved in lesion formation.