毕业典礼 发表于 2025-3-23 12:59:17
http://reply.papertrans.cn/16/1590/158983/158983_11.pngexhilaration 发表于 2025-3-23 15:49:35
http://reply.papertrans.cn/16/1590/158983/158983_12.pngConcomitant 发表于 2025-3-23 21:25:39
http://reply.papertrans.cn/16/1590/158983/158983_13.pngCriteria 发表于 2025-3-23 22:37:36
,Role of Apolipoprotein E in Alzheimer’s Disease: Clues from its Structure,eisgraber 1994). Early studies had connected apoE to nerve regeneration following injury, where apoE participated in local capture and reutilization of neuronal membrane lipids at the injury site during the degeneration and regeneration processes (Boyles et al. 1989; Pitas et al. 1987; Skene and ShoAlveolar-Bone 发表于 2025-3-24 03:15:22
,Apolipoprotein E4, Cholinergic Integrity, Synaptic Plasticity and Alzheimer’s Disease,t in relation to the function of the cholinergic system, which relies to a certain extent on the integrity of phospholipid homeostasis in neurons. Recent evidence indicates that the apoE4 allele has a direct impact on cholinergic function and on cholinomimetic drug response in AD subjects.创造性 发表于 2025-3-24 09:00:20
http://reply.papertrans.cn/16/1590/158983/158983_16.png安抚 发表于 2025-3-24 11:40:01
Apolipoprotein E: Structure, Function, and Possible Roles in Modulating Neurite Extension and Cytos4 without lipid to the dorsal root ganglion or Neuro-2a cells had no effect on neurite outgrowth. The apoE4-mediated effects on inhibition of neurite extension require lipoprotein receptor binding and internalization to elicit the response, and the receptor responsible for the apoE4 effect has beenconstitutional 发表于 2025-3-24 15:39:44
http://reply.papertrans.cn/16/1590/158983/158983_18.pngfaculty 发表于 2025-3-24 23:03:45
http://reply.papertrans.cn/16/1590/158983/158983_19.png愚笨 发表于 2025-3-25 03:00:26
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