Racket
发表于 2025-3-21 19:34:34
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形状
发表于 2025-3-21 23:49:57
Modulating Cholinergic Neurotransmission Through Transgenic Overexpression of Human Cholinesterasescholinergic synapses of . tadpoles and mice. Cholinesterase over-expression should create a local deficit of acetylcholine at the “engineered” synapses and initiate feedback responses from which one may learn about the role of cholinergic neurotransmission in regulating synaptic structure and function.
浪费物质
发表于 2025-3-22 03:15:04
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善辩
发表于 2025-3-22 07:53:42
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送秋波
发表于 2025-3-22 10:24:18
Die Bankkalkulation in der Praxischolinergic synapses of . tadpoles and mice. Cholinesterase over-expression should create a local deficit of acetylcholine at the “engineered” synapses and initiate feedback responses from which one may learn about the role of cholinergic neurotransmission in regulating synaptic structure and function.
nettle
发表于 2025-3-22 15:50:25
https://doi.org/10.1007/978-3-322-98662-7 of ACh (Quinn, 1987). A second, ‘peripheral’ anionic site is so named since it is distant from the active site (Taylor and Lappi, 1975). Bisquaternary AChE inhibitors derive their enhanced potency, relative to homologous monoquatemary ligands (Main, 1976), from their ability to span these two `anionic’ sites, which are .. 14 À apart.
鄙视读作
发表于 2025-3-22 20:59:46
Conference proceedings 1994 metabolic effects that are isoform specific. Isoform-specific interactions of apoE therefore become critical in the mechanism of AD pathogenesis. Detailed characterization of the binding of the apoE isoforms with proteins and peptides relevant to the pathology of the disease may be critical in unde
不能平静
发表于 2025-3-22 21:40:20
ay involve metabolic effects that are isoform specific. Isoform-specific interactions of apoE therefore become critical in the mechanism of AD pathogenesis. Detailed characterization of the binding of the apoE isoforms with proteins and peptides relevant to the pathology of the disease may be critic
effrontery
发表于 2025-3-23 05:06:30
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急急忙忙
发表于 2025-3-23 06:04:58
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