泥土谦卑
发表于 2025-3-23 13:08:29
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AND
发表于 2025-3-23 15:59:36
Huntington disease: new insights on the role of huntingtin cleavage,ragments into the nucleus of striatal neurons. Increasing nuclear localization of huntingtin N-terminal fragments within medium spiny neurons of the striatum occurs concomitantly with the onset of selective neurodegeneration. Because huntingtin is a caspase substrate and truncated huntingtin fragmen
Collected
发表于 2025-3-23 18:00:47
,Autosomal-dominantly inherited forms of Parkinson’s disease,ce and typical Lewy-body pathology (PARK 1), and mutations have been identified in the gene for α-synuclein in these kindreds. Two other loci in families with dominant inheritance have been mapped, to chromosome 2p13 (PARK 3) and to chromosome 4p, respectively. A gene causing autosomal recessive par
hematuria
发表于 2025-3-23 23:43:07
,Subcellular localization of α-synuclein in primary neuronal cultures: effect of missense mutations,n appears to aggregate into distinct punctate inclusions in the cytoplasm and proximal neurites. Unlike Lewy bodies, these structures are not ubiquitin positive. These regions of α-synuclein accumulation are observed following transfections with wild-type, Ala30Pro or Ala53Thr α-synuclein; neither m
戏法
发表于 2025-3-24 04:42:16
Iron chelating, antioxidant and cytoprotective properties of dopamine receptor agonist; apomorphinehat continuous sustained stimulation of dopamine receptor may be responsible for its unusual pharmacological activity. Apomorphine has a far more broad neuroprotective activity in the various models as compared with 1-selegiline and may therefore be an ideal drug to study neuroprotection in parkinso
命令变成大炮
发表于 2025-3-24 08:08:01
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white-matter
发表于 2025-3-24 13:59:15
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indubitable
发表于 2025-3-24 18:43:38
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invulnerable
发表于 2025-3-24 20:46:11
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Influx
发表于 2025-3-25 00:16:43
Mechanisms of tissue injury in multiple sclerosis: opportunities for neuroprotective therapy,of therapeutic delivery systems, which would reach the site of disease activity within the CNS, will permit the administration of inhibitors either of the cell death pathway or of effector target inter action and opens new avenues to neuroprotection approach.