体贴
发表于 2025-3-26 22:51:45
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思考才皱眉
发表于 2025-3-27 04:22:13
Filomena Maggino,Silvana Schifini D’Andreae. Antisynthetase syndrome is characterized by the presence of anti-tRNA synthetase autoantibodies, myositis, interstitial lung disease, arthralgia, mechanic’s hands, and Raynaud‘s phenomenon. Here, we describe a case of antisynthetase syndrome associated with anti-Jo-1 autoantibody to illustrate the clinical and muscle biopsy features.
弄皱
发表于 2025-3-27 07:03:25
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anachronistic
发表于 2025-3-27 12:47:43
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分散
发表于 2025-3-27 15:49:55
Chandra Hawley Orrill,Rachael Eriksen Brownifest fixed proximal limb weakness, cardiac conduction block, cardiomyopathy, hearing loss, retinopathy, and diabetes mellitus, among others. Here we present a case of mitochondrial myopathy with clinical, pathological, and genetic features consistent with Kearns-Sayre syndrome.
airborne
发表于 2025-3-27 19:55:26
Book 2020etation. Divided into three parts, the book begins with introductory chapters that review muscle, nerve, and skin biopsy indications and procedures, normal muscle and nerve histology, and common muscle and nerve pathology. The 28 myopathy case chapters in Part 2 and 11 neuropathy case chapters in Pa
某人
发表于 2025-3-27 23:39:53
Wim Groot,Henriëtte Maassen van den Brinkin skeletal muscle. The chapter concludes with a summary of the wide range of morphological changes that can be seen in skeletal muscle, including brief comments about some of the more common artifacts encountered in the interpretation of muscle biopsies.
清澈
发表于 2025-3-28 04:09:18
Renato G. Flôres Jr.,Bruno B. Rocheutaryl-coenzyme A reductase (HMGCR) or anti-signal recognition particle (SRP) autoantibody in a subset of patients. Here we present a case of immune-mediated necrotizing myopathy associated with anti-SRP autoantibody, which is severe and refractory to immunosuppressive therapy.
Somber
发表于 2025-3-28 08:10:00
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IST
发表于 2025-3-28 10:46:40
M. Joseph Sirgy,Don Rahtz,A. Coskun Samliion of primary endomysial inflammation, rimmed vacuoles, tubulofilamentous inclusions, excessive cytochrome c oxidase (COX)-deficient myofibers, and diffuse upregulation of class I major histocompatibility complex (MHC1). Here we present a case of IBM to illustrate the clinical and pathological features.