Intimidate
发表于 2025-3-21 17:31:24
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babble
发表于 2025-3-21 23:17:47
https://doi.org/10.1007/978-3-031-01666-0inely as predictive markers for tumor response to anti-hormone therapy. However, about 30 % of patients with positive receptor status (expected to benefit from anti-hormone treatment) will face a therapeutic failure, showing the limitations of these markers.
乞丐
发表于 2025-3-22 01:58:56
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Alveoli
发表于 2025-3-22 08:22:51
Heterogeneous Buffering for 3D NoCsne, or, in fact, predecessors of this famed ‘guardian of the genome’? . studies clearly reveal distinct physiological roles for p53, p63, and p73. But do these belie cooperative or antagonistic interactions within the p53 gene family?
笨重
发表于 2025-3-22 10:55:15
https://doi.org/10.1007/978-3-030-96794-9as not been demonstrated, and p53 tumor suppressor activity may not require the acute p53 response to DNA damage (Komarov et al., 1999). Nonetheless, the genotoxic chemicals and ionizing radiation that are clinically used to treat human cancer indisputably activate wild type p53.
一瞥
发表于 2025-3-22 13:03:23
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沙发
发表于 2025-3-22 20:09:24
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警告
发表于 2025-3-22 23:17:45
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Ccu106
发表于 2025-3-23 01:29:49
Book 2005of p53 biology or deregulation are scattered in many different publications, it is extremely difficult to access the full scale of relevant information of any specific p53-related topic. Review arcticles, despite their fundamental role in disseminating knowledge, usually focus only on general mechan
清唱剧
发表于 2025-3-23 06:39:24
https://doi.org/10.1007/978-3-030-96794-9 genetics in cancer research. I therefore arranged to visit Arnie’s lab to learn more about his research program. We discussed many topics, but not about how the large transforming protein (T antigen) of SV40 (SV40TAg) interacted with a putative ~54kDα cellular protein (Linzer and Levine, 1979).