BET 发表于 2025-3-26 23:53:13
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Front Matter post-graduates, post-doctoral researchers and research scientists who are interested in expanding their knowledge of DNA nanotechnology. It provides readers an impression of the latest developments in this exciting filed..978-3-642-42682-7978-3-642-36077-0草率女 发表于 2025-3-27 08:50:37
interaction. The results of these studies suggest that genetic liability to CHD is influenced significantly by a locus or loci in the apo AI/CIII/AIV gene region. Since the association of this region with familial CHD involves different alleles or haplotypes in different sections of the population (哀求 发表于 2025-3-27 12:32:42
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Erkenntnisse empirischer Forschung und pädagogischer Praxis zu einzelnen thematischen Feldern von Ab TTD clinical symptoms except for clinical and cellular photosensitivity that, however, in TTD does not result in increased carcinogenesis. The recent finding that alterations in the β-subunit of the basal transcription factor TFIIE result in the non-photosensitive form of TTD highlights the relevan橡子 发表于 2025-3-27 20:40:22
for up to 96 hours after treatment, compared with the rat liver extracts, where active enzyme was detectable from 24 hours and by 96 hours an increase in enzyme levels compared to control values was observed. This lack of active O.-methylguanine-DNA methyltransferase explains the apparent stabilitytenosynovitis 发表于 2025-3-28 01:14:02
Inhaltliche und methodische Grundlagenoth transcription and NER. . and . encode two subunits of TFIIH, and also TTDA has been recendy identified as a new component of the TFIIH complex. The discovery of this unexpected link between DNA repair and transcription was crucial to rationalize the TTD pathological phenotype as well as the puzz木质 发表于 2025-3-28 02:48:17
Empirische Befundehe mutant .. was found to be unable to process lesions induced by the crosslinking agent nitrogen mustard (HN2) into point mutations (measured as sex-linked recessive lethals). Alkylation damage leads to increased point mutation frequencies in the excision repair deficient mutant .., but to reduced